Lupus News Today reports on a study recently published in Science which suggested excessive production of type I interferon (IFN-I) in people with systemic lupus erythematosus (SLE) may be caused by small fragments of mitochondrial DNA that is released upon damage.

Mary K. Crow, MD, physician-in-chief and chief of rheumatology at HSS, published an accompanying perspective article on the findings and stated, “There is progress in developing therapies to inhibit interferon’s impact on the immune system and improve outcomes for patients with lupus.”

Read the article at Lupusnewstoday.com.